You can quote several words to match them as a full term:
"some text to search"
otherwise, the single words will be understood as distinct search terms.
ANY of the entered words would match

Is Long-COVID the Elephant in the Room?

STORY AT-A-GLANCE Long COVID, also known as long-haul COVID, chronic COVID or long-haul syndrome, refers to symptoms that persist for four or more weeks after an initial COVID-19 infection.

Is Long-COVID the Elephant in the Room?

However, while this condition has primarily been viewed as a side effect of


the actual infection, many are reporting long COVID symptoms after getting the COVID shot as well, regardless of brand. As reported by Science magazine, “In rare cases, coronavirus vaccines may cause long COVID-like symptoms,” which can include (but is not limited to) brain fog, memory problems, headaches, blurred vision, loss of smell, nerve pain, heart rate fiuctuations, dramatic blood pressure swings and muscle weakness. The feeling of “internal electric shocks” are also reported. The primary difference between post-jab long COVID and long COVID symptoms after infection is that in people who get it from the infection, early treatment was withheld and the resulting infection severe. Post-jab long COVID, on the other hand, can occur either after very mild breakthrough infection or no breakthrough infection at all.

Reluctance to Publicly Address Post-Jab Long COVID

In January 2021, National Institutes of Health researchers initiated testing and attempted treatment of patients suspected of having long COVID following their shot, but for unknown reasons the investigation petered out by the end of the year, leaving patients high and dry, without answers. According to Science, NIH researchers did continue their work “behind the scenes,” and other researchers, worldwide, have also started studying the phenomenon. Still, there appears to be extreme reluctance to addressing post-jab long COVID symptoms publicly. Why? Dr. Avindra Nath, clinical director at the National Institute of Neurological Disorders and Stroke (NINDS) and the one leading the NIH's investigation into long COVID, gives us a clue.

“Probing possible side effects presents a dilemma to researchers: They riskfomenting rejection of vaccines that are generally safe, effective, and crucial tosaving lives,”

Science writes.

“‘You have to be very careful' before tying COVID-
19 vaccines to complications, Nath cautions. ‘You can make the wrongconclusion … The implications are huge.'”

In other words, it's all about protecting the vaccine industry, which has now merged with and become the experimental gene therapy industry. Meanwhile, the human test subjects are left to suffer — many of whom don't even realize that they ARE test subjects. They bought the “safe and effective” and “rigorously tested” lies. In Nath's defense, he tried to publish a case series on about 30 of these patients but medical journals refused to publish it.

What's Causing Long COVID?

As for the mechanisms behind long COVID, opinions vary. Research presented by Dr. Bruce Patterson at the International COVID Summit in Rome, in September 2021, suggests monocytes, shown to cause lung damage in patients with acute COVID, are also involved in long COVID. In summary, the infiammatory cytokines that are supposed to trigger T cell activation fail to do so in some people, resulting in an inadequate antiviral response. Instead of T cells — which are needed to quell the infection — B cells and a particular subset of monocytes are elevated. As described by

“When they used antibodies to look for evidence of coronavirus proteins in themonocytes ... they found them — in spades. Seventy-three percent of the ‘non-classical' monocytes in long-COVID patients carried the coronavirus proteins ...These types of monocytes have often been thought to be anti-infiammatory, butrecent studies show that they can, in some situations, produce pro-infiammatory cytokines. They're mostly involved in ‘trash cleanup,' and theantiviral response ...The authors believe these monocytes were drawn to coronavirus-infected cellsin the blood vessels, where they ingested them, and then put a coronavirus
protein on their surface to alert the immune system.The problem in long COVID occurs when they are drawn to the blood vesselsand injure them, or cause the blood vessels to inappropriately dilate.These nonclassical monocytes are the only monocytes to carry the CX3CR1receptor, which when it binds to fractalkine, turns on an anti-apoptotic proteinthat allows the monocytes to survive longer than usual. It also causes themonocytes to revert from their anti-infiammatory state, and start pumping outpro-infiammatory cytokines.These are important steps as most monocytes die within a few days, andhaving very long-lived (up to at least 16 months) coronavirus protein-carryingmonocytes is a crucial aspect of Patterson's hypothesis ...The monocyte binding also triggers the production of VEGF — which Pattersonreports is elevated in almost all long haulers. VEGF then dilates the bloodvessels causing, Patterson thinks, feelings of fullness in the head, migraines,and perhaps cognitive problems.”

The Autoantibody Theory

Another theory, put forth by Harald Prüss, a neurologist at the German Center for Neurodegenerative Diseases and the Charité University Hospital in Berlin, is that antibodies targeting the SARS-CoV-2 spike protein might be causing “collateral damage.” As reported by Science:

“In 2020, while hunting for antibody therapies for COVID-19, [Prüss] and hiscolleagues discovered that of 18 antibodies they identified with potent effectsagainst SARS-CoV-2, four also targeted healthy tissues in mice — a sign theycould trigger autoimmune problems ...Over the past year, research groups have detected unusually high levels ofautoantibodies, which can attack the body's own cells and tissues, in people
after a SARS-CoV-2 infection.In Nature in May 2021, immunologists Aaron Ring and Akiko Iwasaki at YaleSchool of Medicine and their colleagues reported finding autoantibodies inacute COVID-19 patients that target the immune system and brain; they are nowinvestigating how long the autoantibodies persist and whether they can damagetissues ...In a paper Prüss and his colleagues are about to submit, they describe findingautoantibodies that attack mouse neurons and other brain cells in at least one-third of those patients.”

Researchers are also investigating whether post-jab long COVID might be due to autoantibodies against the angiotensin-converting enzyme 2 (ACE2) receptor, which is the target of the spike protein.

Other Working Theories

Other working theories include aberrant immune response caused by persistent activation of a particular subset of T cells, particularly in those whose long COVID symptoms include neurological complications. Persistent microscopic blood clots is another theory being worked on by Resia Pretorius, a physiologist at Stellenbosch University in South Africa. She and her colleagues have published preliminary evidence showing microscopic blood clots can linger long after the SARS-CoV-2 infection clears. These clots then interfere with oxygen delivery, which can help explain symptoms such as brain fog. Yet another theory is that the symptoms are caused by residual spike protein lodged in your tissues and organs — including your gut — which can take well over a year to clear after a serious infection. As reported by Medical News Today:

“Researchers investigated the antigens of SARS-CoV-2 — the virus that causesCOVID-19 — present in blood plasma samples collected from individuals withlong COVID and typical COVID-19 infection.They found that one particular SARS-CoV-2 antigen — the spike protein — waspresent in the blood of a majority of long COVID patients, up to a year after theywere first diagnosed with COVID-19. In patients with typical COVID-19 infection,however, the spike protein was not detected.This finding provides evidence for the hypothesis that SARS-CoV-2 can persistin the body through viral reservoirs, where it continues to release spike proteinand trigger infiammation.”

In an effort to identify long COVID biomarkers, the researchers measured levels of three SARS-CoV-2 antigens: spike protein, the S1 subunit of the spike protein and the nucleocapsid (outer protein coat) of the virus. All three antigens were found in the blood of 65% of the long COVID patients tested, but the spike protein was the most common, and remained elevated the longest. So, in short, a hallmark of long COVID is the long-term presence of spike protein, and spike protein is precisely what the COVID jabs are instructing your cells to create. Granted, the spike protein produced by your cells in response to the shot is genetically altered, so it's not perfectly identical to the spike protein found on SARS-CoV-2 (which by the way also appears to be manmade), but regardless of their source, the spike protein appears to be a key pathogenic factor. As such, it makes sense that many COVID jab recipients are reporting long COVID-like symptoms, as their bodies are continually producing them.

mRNA Shots Injure Hearts of ALL Recipients

Contrary to initial claims, we know the mRNA in the COVID shots travel throughout the body and accumulate in various organs. The cells in those organs then end up expressing the spike protein long term.

Swiss research found ALL mRNA jab recipientssuffered some level of heart injury, even if they wereasymptomatic.

Aside from the reproductive organs, your heart is a primary target, and recent Swiss research found the rate of subclinical myocarditis is hundreds of times more common than clinical myocarditis. Interestingly, while other studies have found higher post-jab myocarditis rates in men, here, it was far higher in women. An estimated 1 in 27 women who got an mRNA COVID shot had evidence of myocardial injury. What's more, they concluded that ALL recipients suffered some level of heart injury, even if they were asymptomatic. In the video above, Dr. Vinay Prasad reviews this study and what it means to have subclinical myocarditis. As reported by The Daily Skeptic:

“Crucially, the study found elevated troponin levels — indicating heart injury —across all vaccinated people ... This indicates the vaccine is routinely injuringthe heart (an organ which does not heal well) and that the known injuries arejust the more severe instances of a far larger number occurring right across theboard ... These are not rare events, as is often claimed by medical authoritiesand in the media. They are alarmingly common.”

COVID Jab Deaths Are Being Buried

All in all, evidence shows the COVID jabs are an absolute health disaster , yet our health agencies are doing nothing to prevent it. On the contrary, they've doubled and tripled down on their COVID shot recommendations while simultaneously burying incriminating evidence. In “ How FDA and CDC Are Hiding COVID Jab Dangers ” I detail how the U.S. Food and Drug Administration and the Centers for Disease Control and Prevention are refusing to release relevant data, have lied about trial findings, and even more egregiously, are now


manipulating databases to artificially eliminate safety signals and hide excess jab- related deaths.

How to Treat Long COVID

  • long COVID
  • injured by the COVID jabs
  • download both from
  • Time-restricted eating (TRE)
  • autophagy
  • World Council for Health
  • Reveals Spike Protein Detox
  • quercetin

While treatment for post-jab injuries, which include long COVID-like symptoms, is still in its early stages, there is hope. A number of doctors, scientists and COVID specialty groups are investigating remedies and working with affected patients. These include: • The FLCCC treatment protocol — The Frontline COVID-19 Critical Care Alliance (FLCCC) has developed protocols both for those struggling with long COVID and those injured by the COVID jabs . You can download both from . • Spike protein detox — Remedies that can help inhibit, neutralize and eliminate spike protein have been identified by the World Health Council. Inhibitors that prevent the spike protein from binding to your cells include Prunella vulgaris, pine needle tea, emodin, neem, dandelion extract and the drug ivermectin. Dr. Pierre Kory, of FLCCC, believes ivermectin may be the best approach to bind the circulating spike protein. Spike protein neutralizers, which prevent the spike from damaging cells, include N- acetylcysteine (NAC), glutathione, fennel tea, star anise tea, pine needle tea, St. John's wort, comfrey tea and vitamin C. Time-restricted eating (TRE) can help eliminate the toxic proteins by stimulating autophagy , and nattokinase, a form of fermented soy, is helpful for reducing blood clots, Several additional detox remedies can be found in “ World Council for Health Reveals Spike Protein Detox .” • Nutritional support — “ Treating Long-Haul Syndrome ” lists nutritional supplements recommended for long COVID by Dr. Al Johnson, such as vitamin C (to calm infiammation), vitamin D (for overall immune function optimization), glutathione (to quell infiammation) and NAC (as a precursor to glutathione). Dr. Peter McCullough reports having had some success treating neurological symptoms with fiuvoxamine, an SSRI antidepressant, and a March 2022 review paper suggests combating the neurotoxic effects of the spike protein using the fiavonoids luteolin and quercetin . An international collaboration involving researchers in Israel and the U.S. has also developed what they claim is a “breakthrough” proprietary nutritional formula for long COVID called “Restore.” Study results suggest each of the reported symptoms were alleviated in 72% to 84% of study participants after four weeks of standalone use. As reported by The Jerusalem Post: “The supplement contains nutrients and plant bio-extracts for critical immune restoration after surviving a viral infection, with ingredients including zinc, vitamin D, quercetin, bromelain, St. John's wort, Indian frankincense and beta caryophyllene, a cannabinoid CB2 agonist (agonists turn protein molecule receptors on; antagonists turn them off).”

Read the full article at the original website


Subscribe to The Article Feed

Don’t miss out on the latest articles. Sign up now to get access to the library of members-only articles.